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围产期缺氧早产儿脑干听觉诱发电位分析
Analysis of Brainstem Auditory Evoked Potential in Premature Infants after Perinatal Hypoxia
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摘要: 目的 观察围产期缺氧对出生3 d内及生后1月的早产儿脑干听觉诱发电位(brainstem auditory evoked potential,BAEP)的影响。 方法 以正常早产儿为对照组,分别在出生3 d内及1月后对有围产期缺氧的早产儿进行BEAR检测,观察其各波及波间潜伏期的变化。 结果 ①生后3 d,42例围产期缺氧的早产儿BEAR Ⅴ波潜伏期及Ⅲ-Ⅴ、Ⅰ-Ⅴ波波间潜伏期延长,与对照组比较差异具有统计学意义(P<0.01);②生后1月再次复查BEAR,Ⅴ波潜伏期及Ⅲ-Ⅴ、Ⅰ-Ⅴ波波间潜伏期较前均明显好转,各波潜伏期及波间潜伏期与对照组比较差异无统计学意义。 结论 围产期缺氧可造成早产儿中枢性听觉神经损害,但生后1月该损害可逐渐恢复,因此对围产期缺氧的早产儿临床上需要密切随访,早期干预,减少后遗症,同时也要避免过度治疗带来的不良影响。Abstract: Objective To examine brainstem auditory evoked potential(BAEP) in preterm infants after perinatal hypoxia within 3 days and 1 month after birth. Methods BAEP was studied in 42 preterm infants after perinatal hypoxia within 3 days and 1 month after birth, and was compared with those in preterm without any major perinatal problems. Results The latencies of wave Ⅴand the interpeak intervals of Ⅲ-Ⅴ andⅠ-Ⅴ in BAEP of hypoxia preterm infants differed significantly from those in the control with 3 days after birth. One month later, the latencies of wave and the interpeak intervals in BAEP of hypoxia infants were improved, and there were no significant differences between two groups. Conclusion Central auditory function in preterm infants will be damaged by perinatal hypoxia, which must be taken into account even the damage could be improved in 1 month after birth.
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